Whether you live in an apartment or house, or you just want to keep your home free of electromagnetic fields, there are a number of ways you can reduce exposure. One of the easiest is to reduce the use of electronic devices. You can also turn to EMF blocker paint to block EMF radiation from reaching your house. Another easy way to protect your house from EMF radiation would be to install an RF shielding cover. It's a kind of net that has EMF shielding and is used to block EMFs from entering a space. Another alternative is to have your home equipped with a conductive enclosure. https://www.pearltrees.com/ferrysingle30/item512698106 are called Faraday cages.
A number of studies have proven how the EMF that is not ionized can cause antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to involve down-regulation of cancer stem cells. This could explain the long-term response seen in some patients with advanced HCC. However, the reason behind AM EMF's impact on cancer patients isn't yet clear.
https://maidease55.werite.net/post/2023/04/13/EMF-Block-Paint-plus-EMF-Shielding from AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were studied in mice. The tumours were divided into three groups. The first group was not exposed RF EMF. Second group members were exposed RF EMF at a frequency that is similar to that of humans. https://mackinnon-potter.hubstack.net/emf-block-paint-plus-emf-shielding-1681421174 were exposed RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on tumours was evaluated against that of RCF. The results indicated that tumours treated with HCCMF showed significant shrinkage. However, tumours treated with RCF did not show any evidence of tumour shrinkage.
The reason for tumour-specific AM RF EMF could be driven by the fact that tumor cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is caused through CACNA1H, a protein which is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H could have more broader implications in the treatment and diagnosis of various cancers.
The tumours in the control group were not exposed to EMF from radiofrequency, and fed a standard mouse diet. The tumors of those in the HCCMF group were infected with Huh7 cells at the time they were between five and seven weeks old. The tumours were then euthanized after they had a high burden.
The tumours in the three groups also showed distinct growth curves. The tumors treated with HCCMF showed a significant decrease in size of the tumor after 8 weeks. However, the tumours which were treated by RCF didn't show signs of shrinkage. The difference was highly significant. The tumors treated by RCF had necrosis, which is common in tumours exposed to RCF. It is possible that the necrosis was caused by the lack of oxygen in the more invasive tumors.
In sum, the results indicate an AM-RF EMF exhibits anticancer effects in vitro and in vivo. A number of studies have proven it is true that AM RF EMF produces measurable reduction in tumours for HCC patients. It is possible that AM RF EMF produces these effects due to CACNA1H which is a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting influence on the growth of HCC tumours in the vivo.